ductal
Cell Sentence Context
Table 1. Analysis of context sentence of goblet tissue in 12 abstracts.
| PMID | Senteces |
|---|---|
| 32311451 | The results reveal that ACE2 mRNA and protein are highly expressed in the small intestinal enterocytes but not in the goblet cells or intestinal immune cells. |
| 32413319 | We identify ACE2 and TMPRSS2 co-expressing cells within lung type II pneumocytes, ileal absorptive enterocytes, and nasal goblet secretory cells. |
| 32816230 | The following cell types expressed ACE2: basal, apical, goblet, minor salivary, and endothelial cells. […] TMPRSS2 was found in goblet and apical respiratory cells. |
| 32946807 | Intestinal goblet cells, enterocytes, and kidney proximal tubule cells appear highly permissive to SARS-CoV-2, consistent with clinical data. |
| 33060413 | The CIC results revealed decreased density and cell size of goblet cells and moderate to high enlargement, squamous changes, and increased nucleocytoplasmic ratio in nongoblet epithelial cells in the COVID-19 group compared with the control group. |
| 33112891 | Single cell RNA-Seq data from trachea indicated positive signals along the respiratory tract in key protective cell types including club, goblet, proliferating, and ciliary epithelial cells; while in lung the ratio of ACE2-expressing cells was low in all cell types (<2.6%), but was highest in vascular endothelial and goblet cells. |
| 33186749 | However, SARS-CoV-2 spared goblet cells lacking ACE2 expression. |
| 33292872 | We show co-expression of ACE2 and TMPRSS2 with nAChRs and α7 nAChR in particular in intestinal stem cells, goblet cells, and enterocytes. |
| 33307034 | Immunohistochemistry and Alcian blue/periodic acid-Schiff staining showed decreased Ki67, increased cleaved caspase 3, and decreased numbers of mucin-containing goblet cells, suggesting that the inflammation induced by these 2 types of inoculation with SARS-CoV-2 impaired the GI barrier and caused severe infections. |
| 33336811 | Viral penetration requires binding of the viral spike (S) protein to a specific cellular receptor (ACE2) highly expressed in a nasal goblet and ciliated cells. |
| 33457378 | The hyperstimulation of tracheal goblet cells secreting mucus is 1 pathophysiological hypothesis. […] Painless, non-inflammatory tracheal hypersecretion, which has yet to be described in post-acute COVID-19, may be an additional long-term symptom of the disease.The hyperstimulation of mucus-secreting tracheal goblet cells by the coronavirus, leading to tracheal discomfort and a feeling of suffocation, is 1 pathophysiological hypothesis. |
| 33572760 | Mucociliary defense, mediated by the ciliated and goblet cells, is fundamental to respiratory fitness. |